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Various central and peripheral nervous system disorders may produce camptocormia (from the Greek: “bent spine”), truncal flexion while standing and walking, which abates in sitting or recumbent positions.1 In a recent review of patients with camptocormia evaluated and treated over the last 25 years, levodopa did not improve the truncal flexion accompanying ambulation of any patients with dystonia or Parkinson disease.1 Additionally, “sensory tricks” were exhibited by their patients with camptocormia secondary to axial dystonia.1 Sensory tricks are heterogeneous maneuvers, often tactile or proprioceptive, which alleviate abnormal postures and are characteristic of organic dystonias.2 In this report, I discuss a 5-year experience with a middle-aged man with apparent dopa-responsive dystonic camptocormia, possibly a forme fruste of dopa-responsive dystonia (DRD) and an intriguing sensory trick.
In 1999, a previously healthy 48-year-old man …
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- Dopa-responsive dystonic camptocormia
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